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Monoubiquitination typically modulates protein activity, localization, interaction partners, and endocytic trafficking [68], whereas K63-linked polyubiquitination mediates signal transduction pathways and K48-linked polyubiquitination targets substrates for proteasomal degradation [66, 67]
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Ubiquitin ligase (MARCH 1) can reduce the number of receptors on the surface of cells by ubiquitinating INSR, and its expression in the white adipose tissue (WAT) of obese individuals is increased, further exacerbating receptor defects (81)
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