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Its regulation centers on four interconnected processes: iron metabolism, polyunsaturated fatty acids (PUFA) peroxidation, antioxidant defense failure, and mitochondrial dysfunction ( 2.1 Iron metabolism and ferroptosis Iron is central to ferroptosis, as its redox activity catalyzes the formation of reactive oxygen species (ROS) via Fenton reactions, triggering cellular membranes damage and cell death ( A critical regulator of intracellular iron levels is ferritinophagy, the selective autophagic degradation of ferritin mediated by nuclear receptor coactivator 4 (NCOA4)

With the rich abundance of mitochondria, kidney is an organ with a high consumption of energy

found that histidine catabolism affects the sensitivity of cells to methotrexate, and dietary supplementation of histidine maintains a suitable chemotherapy effect with a low dose of medication

Fller M, Feil S, Ghoreschi K, Koka S, Gerling A, Thunemann M, et al