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However, under normal physiological conditions, these alternative mechanisms of regulating cell death largely remain unknown ( However, there are still gaps in our understanding of the triggering, execution, and propagation mechanisms of ferroptosis in kidney diseases that require further research

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Due to its key role in metabolism and detoxification processes it continuously deals with different substances and compounds resulted from metabolism paths which may have destructive impacts on it [1]

Our results revealed that SFN treatment considerably inhibited the phosphorylation of STAT3 and the expression of K16 and K17 (Fig